Dr. Blaylock Reports #7; Atherosclerosis,  Failure of Statins; Complications; Molecular Basis of Plaque formation.  Immunoexcitotoxicity episode artwork

EPISODE · Feb 7, 2024 · 47 MIN

Dr. Blaylock Reports #7; Atherosclerosis, Failure of Statins; Complications; Molecular Basis of Plaque formation. Immunoexcitotoxicity

from Dr. Blaylock Reports

SUMMARY: Dr. Blaylock Reports provides the data indicating Cholesterol is not involved in atherosclerotic placque formation. Statin drugs promoted by the pharmaceutical industry produce muscle weakness, memory loss, immune suppression while being of no value in stopping plaque formation. Statins also lower Cholesterol and Co. Q10 levels. Co -Q10 is necessary for muscle function. Giving high doses of Omega 6 Fatty acids, are excitatory to cells, and will produce plaque formation in animal studies. Measurements of LDL, HDL are useless without detailed subgroup analysis. Placques result from an endothelial tear, an inflammatory response at the injury site, macrophage accumulation (inflammatory response) and absorption of Omega 6 FA to form foam cells. This immunologic response is followed by an excitatory reaction aggravated by ingested glutamate to accelerate this plaque formation. Ultimately rupture of the placque produces a myocardial or cerebral infarction. Atherosclerosis is an inflammatory disease, best treated by a diet low in excitatory fatty acids and other molecules, Mediterranean Diet, Fish, fruit and vegetable ingestion. With a 50% obesity rating world wide, statin treatment has not made a difference. 40 minutes. Excellent (JIA)

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Dr. Blaylock Reports #7; Atherosclerosis, Failure of Statins; Complications; Molecular Basis of Plaque formation. Immunoexcitotoxicity

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