Episode 321 - Mike Beckstead, PhD episode artwork

EPISODE · Dec 4, 2025 · 41 MIN

Episode 321 - Mike Beckstead, PhD

from Neuroscientists Talk Shop · host Neuroscientists Talk Shop

On December 4, 2025 we talked with Dr. Mike Beckstead about his work on changes in dopaminergic neuron excitability in the ventral tegmental area in an animal model of Alzheimer's disease. Mike explained the molecular mechanism of the excitability change and the change in gene expression at its cause.Guest: Mike Beckstead, Professor and Hille Family Foundation Chair in Neurodegenerative Disease Research, Aging & Metabolism Research Program Oklahoma Medical Research FoundationParticipating:Matt Wanat, Department of Neuroscience, Developmental and Regenerative Biology, UTSAHost:Charles Wilson, Department of Neuroscience, Developmental and Regenerative Biology, UTSAThanks to James Tepper for original music

Episode metadata supplied by the publisher feed · Published Dec 4, 2025

On December 4, 2025 we talked with Dr. Mike Beckstead about his work on changes in dopaminergic neuron excitability in the ventral tegmental area in an animal model of Alzheimer's disease. Mike explained the molecular mechanism of the excitability change and the change in gene expression at its cause.Guest: Mike Beckstead, Professor and Hille Family Foundation Chair in Neurodegenerative Disease Research, Aging & Metabolism Research Program Oklahoma Medical Research FoundationParticipating:Matt Wanat, Department of Neuroscience, Developmental and Regenerative Biology, UTSAHost:Charles Wilson, Department of Neuroscience, Developmental and Regenerative Biology, UTSAThanks to James Tepper for original music

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Episode 321 - Mike Beckstead, PhD

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This episode is 41 minutes long.

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This episode was published on December 4, 2025.

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On December 4, 2025 we talked with Dr. Mike Beckstead about his work on changes in dopaminergic neuron excitability in the ventral tegmental area in an animal model of Alzheimer's disease. Mike explained the molecular mechanism of the excitability...

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