Механизмы потери костной массы при хроническом стрессе и дисфункции HPA-оси

Хронический стресс вызывает потерю костной массы через дисфункцию оси HPA и нарушение транспорта рецепторов глюкокортикоидов (GR). Рост уровня FKBP5 и 11β-HSD1 блокирует работу GR, вызывая воспаление и гибель остеобластов, что ведет к прогрессирующему остеопорозу.

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