Skip to content
PALS | Pediatric Shock Extended episode artwork

Sep 14, 2026 45 min

PALS | Pediatric Shock Extended

from STAT Stitch Deep Dive Podcast Beyond The Bedside

. Core Physiology & Definition Definition: Shock is inadequate tissue perfusion to meet metabolic demands and oxygenation needs1. Hypotension is not required for diagnosis; shock exists with normal, high, or low SBP1. Pediatric Focus: Infants have fixed stroke volume and depend on elevated heart rate to maintain CO3. Stroke Volume: Preload (volume), Contractility (pump force), and Afterload (resistance)34. 2. Severity & Compensatory Continuum Compensated Shock: Body mechanisms (tachycardia, elevated SVR, venoconstriction) maintain normal SBP despite hypoperfusion56. Key signs: tachycardia, delayed refill, cool skin, oliguria67. Hypotensive Shock: Compensatory mechanisms fail, causing low SBP and imminent arrest7. Hypotension is a late finding in most shock types, except septic shock where SVR drops early7. Progression: Compensated shock can last hours, but hypotensive shock deteriorates in minutes8. 3. The 4 Shock Types: Differential Pattern Recognition A. Hypovolemic Shock (Most Common) Mechanism: Intravascular volume loss (fluid loss) causing low preload and CO9. Signs: "Quiet tachypnea" (rapid breathing without retractions), narrow pulse pressure, pale/cool skin, delayed refill, weak pulses910. B. Distributive Shock Mechanism: Vasodilation causing blood maldistribution and relative hypovolemia110. Septic Shock: Commonest form11. Warm Shock: Low SVR, bounding pulses, flushed skin, wide pulse pressure1213. Cold Shock: High SVR, weak pulses, cool skin1213. Anaphylactic Shock: Histamine release causes bronchospasm, upper airway edema (stridor), hives, vasodilation14. Neurogenic Shock: High spinal injury causes loss of sympathetic tone hypotension without tachycardia (normal HR or bradycardia)15. C. Cardiogenic Shock Mechanism: Primary pump failure with low contractility, high afterload. Signs: Tachypnea with increased work of breathing (retractions, grunting, crackles), hepatomegaly, cyanosis1617. D. Obstructive Shock Mechanism: Physical restriction of venous return or cardiac outflow18. Tension Pneumothorax: Tracheal deviation, hyperresonance, absent breath sounds19. Cardiac Tamponade: Muffled heart sounds, pulsus paradoxus (>10 mmHg SBP drop on inspiration)20. Ductal Lesions: Neonatal presentation with differential pre/post-ductal SBP/SpO221. Massive PE: Systemic venous congestion and RV failure22.

Episode details come from the podcast's RSS feed.

Embed this episode

Ready to play

PALS | Pediatric Shock Extended

0:00 45:00

No transcript yet.

Requests help us decide what to transcribe next.

Frequently asked questions

How long is this episode of STAT Stitch Deep Dive Podcast Beyond The Bedside?

This episode is 45 minutes long.

When was this STAT Stitch Deep Dive Podcast Beyond The Bedside episode published?

This episode was published on September 14, 2026.

Can I download this STAT Stitch Deep Dive Podcast Beyond The Bedside episode?

Yes. Use the download control on the episode player to save the publisher-provided media file.